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Wednesday, October 5, 2016


A young boy suffering joint hemophilia

A young boy suffering joint hemophilia

HIV-antibodies in hemophilia patients were already shown in the USA by 1978. The spread did occur remarkably quickly, but those infected, only about one-tenth developed full Aids, picture compared with other HIV-infected persons.

Curiously, the Centers for Diseases Control, for example, even considered it a possible in 1984 that these on average particularly susceptible people who rendered to die at a very early stage, could produce some resistance to the HIV effect.

Indeed, 50-80% of hemophilia patients showed HIV-antibodies in the USA, Denmark, France, Germany, and Britain, but only in approximately 24% of these cases was possible to cultivate viruses from their blood.

Even though the bottles with the factor concentrates are available, it has been impossible till now to cultivate the virus from any of these. Those viruses, which were obtained by special reproductive techniques from hemophilia patients who had recent HIV antibodies were inactive and harmless.

In experiments, it was impossible for them to be transmitted in a cell-free state. Some also showed a further amazing characteristic. Of the virus, samples examined  from a suspect batch from infected hemophilia patients  in one place, five were 100% identical, while two were slightly different.

48% of their structure was different compared with that of other HIV-strains. Hemophilia patients with HIV antibodies hardly ever have Kaposi's sarcoma and rarely HIV or opportunistic agents in the brain. 

Their HI viruses obviously rarely have the necessary alignment on cerebral cells which is needed for Aids. Therefore, there is no increase in cortisol.


Deliberate infection would explain the most 100% genetic conformity seen in the only investigated case. Since there has been no evidence of HIV in the factor concentrates, then the umpteen thousands of infections must have been introduced individually. This would appear impossible.

Are HIV-EIAV-type viruses contained as congenital viruses in the gametes hemophilia patients? Transmission of endogenic retrovirus parts from the mother to the child comes to question. These were activated for example, by factor concentrates.

This could happen through parvoviruses which could be found in the blood concentrates for hemophilia patients. Parviruses are minute viruses, which are particularly resistant. The HI virus type in hemophilia patients is not active without these supplies as well as the well-known HIV-types.

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